One is to add
lactase to the milk, which pre-digests the sugar into glucose and galactose.
Previous studies have shown that 40% babies presented with infant colic suffered from
lactase enzyme deficiency,6,7 an intestinal enzyme that breaks down the lactose into simple sugar (galactose and glucose) in small intestine.8
Effect of a single dose of
lactase on symptoms and expired hydrogen after lactose challenge in lactose-intolerant subjects.
European
lactase persistence genotype shows evidence of association with increase in body mass index.
Predictive values of
lactase activity are lower, probably due to the presence of primary
lactase deficiency in coeliac patients.
A
lactase deficiency does not allow lactose to be broken into absorbable nutrients within the intestinal tract.
Lactase deficiency is largely inherited (congenital and familial deficiency), though a condition known as secondary
lactase deficiency can result from inadequate
lactase production and can be caused by a problem with the small intestine, such as surgery, or another condition, such as Crohn's disease, ulcerative colitis, chemotherapy, celiac disease, or gastroenteritis.
Lactose intolerance, in which the body does not produce enough
lactase (the enzyme that breaks down lactose--milk sugar), may benefit from
lactase supplementation.
INTRODUCTION: Adult type hypolactasia develops due to decreased activity of the enzyme
lactase phlorizin hydrolase and stops its synthesis in mammals after the weaning period.
Lactose intolerance, caused by your small intestine being unable to break down
lactase, an enzyme found in dairy foods.
Standard cow's milk contains a fair amount of lactose, a sugar that gives milk its sweetness and that must be broken down by
lactase, a digestive enzyme produced in the small intestine of most mammals.