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Related to foam cell: Closed cell foam

cell

an independent team of operatives who work together in a CELLULAR MANUFACTURING production environment.
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The macrophage foam cell as a target for therapeutic intervention.
TREM-1 is a positive regulator of TNF-[alpha] and IL-8 production in U937 foam cells
Vitamin D suppressed foam cell formation by reducing LDL cholesterol uptake in diabetic subjects only.
Vitamin D for heart health in type 2 diabetes
Scavenger receptors mediate the uptake of modified lipoprotein particles and promote the development of foam cells. Macrophage foam cells are a source of mediators, including other cytokines and effector molecules, such as hypochlorus acid, superoxide anion, and matrix metalloproteinases.
The atherosclerotic plaque
Macrophages play a key role in atherogenesis by releasing proinflammatory cytokines and forming foam cells in subendothelial lesions.
Induction of proinflammatory cytokines and C-reactive protein in human macrophage cell line U937 exposed to air pollution particulates
Lipid droplet formation induced by a snake venom GIIA [sPLA.sub.2] in macrophages depends on lipid metabolism factors mainly PPAR-[gamma] and PPAR-ft/8, providing new insights into mechanisms of macrophage foam cell formation.
A Snake Venom-Secreted Phospholipase [A.sub.2] Induces Foam Cell Formation Depending on the Activation of Factors Involved in Lipid Homeostasis
However, uptake of oxidized LDL by macrophages causes intracellular accumulation of cholesterol and precipitates their transformation to foam cells. Foam cells produce several proteinases, which degrade extracellular matrices, thereby increasing the fragility of plaques [19].
[[sup.18]F]FDG Uptake in the Aortic Wall Smooth Muscle of Atherosclerotic Plaques in the Simian Atherosclerosis Model
In the present study, we aimed to evaluate whether combination therapy with SGLT2i and DPP-4i was superior to monotherapy with either an inhibitor alone in suppressing macrophage foam cell formation, a critical process in atherosclerosis, or the expression of genes involved in foam cell formation in db/db mice, a model of type 2 diabetes.
Combination Therapy with a Sodium-Glucose Cotransporter 2 Inhibitor and a Dipeptidyl Peptidase-4 Inhibitor Additively Suppresses Macrophage Foam Cell Formation and Atherosclerosis in Diabetic Mice
Punicalagin protects macrophage cells from lipid accumulation and foam cell formation [3-5].
Punicalagin Induces Serum Low-Density Lipoprotein Influx to Macrophages
This material forms a uniform foam cell size where at least 90% of the foam cells are 50 micrometers or less.
Uniform fluoropolymer foams
As anticipated, airflow, foam cell structure and surface appearance, hardness, ball rebound and compression sets were all identical to those seen with the conventional surfactants in Formulation A.
Reducing silicone surfactant emissions in automotive flexible molded foam
It has been reported that oxidatively modified low-density lipoprotein (Ox-LDL) involvement with vascular endothelial growth factor (VEGF) and foam cell formation play an important role in atherosclerosis (AS).
Inhibitions of vascular endothelial growth factor expression and foam cell formation by EGb 761, a special extract of Ginkgo biloba, in oxidatively modified low-density lipoprotein-induced human THP-1 monocytes cells
Chiamydial endotoxins, much less virulent than those of enterobacteriaceae (e.g., Escherichia coli), can promote macrophage foam cell formation in vitro (8,39).
Potential infectious etiologies of atherosclerosis: a multifactorial perspective. (Synopses)

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