So far, most groups have observed NP uptake in the syncytiotrophblast after ex vivo human placental perfusion even if NP translocation to the fetal circulation
was absent or below the detection limit (Menjoge et al.
The result of this fetal circulation
arrangement is that blood sampled from the umbilical arteries will be deoxygenated blood and reflect the metabolic state of the fetus and blood sampled from the umbilical vein will be oxygenated blood from the placenta reflecting the functional aspects of placental gas exchange.
Relation of ANP and BNP to their N-terminal fragments in fetal circulation
: evidence for enhanced neutral endopeptidase activity and resistance of BNP to neutral endopeptidase in the fetus.
This study introduced the experience of EXIT procedure to solve the problems of fetal circulation
induced by a fetal cardiac tumor.
The aims and objectives of the present study includes (i) To detect any abnormalities in fetoplacental unit and fetal circulation
in high risk pregnancies.
On the other hand, when maternal or fetal unconjugated bilirubin levels exceed the maternal capacity for catabolism, the excess bilirubin is transferred to the fetal circulation
and may accumulate in fetal organs.
The transfer of substances and the amount that enters the fetal circulation
are tightly controlled by at least four different mechanisms; simple diffusion, active transport, biotransformation through metabolic enzymes, and phagocytosis or pinocytosis (Syme et al.
"Zolpidem appears to rapidly clear the fetal circulation
; however, if delivery occurs less than 11 hours after the last maternal dose, Zolpidem may still be present in the neonatal circulation."
This, compromised by the substance in the study, presents diminished blood flow with consequently less arrival of nutrients, oxygen and other elements of fetal circulation
, resulting in deficiency of growth and fetal weight.
The principal clinical application of NO has been in the reversal of Persistent Fetal Circulation
. The mechanism of vasodilatation has been well documented.
In a previous study we showed that >50% of the NRBCs in the fetal circulation
and cord blood (i.e., before exposure of these cells to the maternal circulation) are TUNEL positive (11).
Most upper-limb deficiencies are the result of developmental accidents of unknown etiology although theories often focus on disruptions of placental or fetal circulation
. Genetic mutations are the second most common cause.