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* Recognize the clinical manifestations of serotonin syndrome, which may include agitation, diaphoresis, hyperthermia, hyperreflexia, and clonus.
Hypertension, tachycardia, tachypnea, diaphoresis, and dystonia are the most commonly identified symptoms of PSH.
A PSH episode was defined as a cluster of symptoms, which include hyperthermia without a source of infection, hypertension, tachycardia, tachypnea (based on age-appropriate vital sign parameters), diaphoresis, and/or dystonia.
Given the dizziness and diaphoresis initially, patient had an ECG performed which showed lateral ST segment elevation (Figure 1) and had a subsequent troponin I that was positive, 0.49ng/mL, with a Creatine Phosphokinase (CPK) of 617 U/L.
He stopped all antihypertensive medications and had no palpitations, diaphoresis, flushing, headaches, or other symptoms of catecholamine excess.
A 32-year-old National Guard member stationed in Nevada, with no significant past medical history presented with three weeks of progressive dyspnea, fevers, fatigue, diaphoresis, and weight loss.
However, features such as diaphoresis, rigidity, and elevated CK levels are not typical for this syndrome.
The most common clinical manifestation was respiratory distress (50%) followed by neck muscle weakness/ fasciculations (44%), miosis (40%) and diaphoresis (30%).
It might also cause pruritus (16%), urticaria (5-18%), and diaphoresis (3%).
He had decreased level of consciousness, muscle rigidity, diaphoresis, fever, drooling, urinary incontinence, and high blood pressure.
We present a case of sporadic insulinoma in a yo ung, 24-year-old female patient, who presented witl a 2-month history of episodic shaking, diaphoresis increased hunger, confusion, obtundation and fainting Symptoms of neuroglycopenia were predominant, sc the family members reported that the patient has un dergone a personality change.
It inhibits a2 receptors and induces excretion of epinephrine, norepinephrine, and their metabolites; it also increases in hydrocortisone and corticotrophin-releasing factor.[sup][3] Alcohol inhibits the excitatory neurotransmitter glutamate by suppression of postsynaptic-methyl-d-aspartate glutamate receptor.[sup][4] Alcohol reinforces the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) at GABA-A receptors.[sup][3] After prolonged intake of alcohol, reductions lead to CNS hyperexcitation such as tachycardia, hypertension, diaphoresis insomnia, anxiety, increased BT, as well as hand tremor.