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1. The principal of a bond. For example, securities dealers create zero-coupon Treasury receipts by purchasing a regular Treasury bond and separating the interest coupons from the corpus. See also coupon stripping.
2. The principal amount of an estate or trust.
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References in periodicals archive ?
Chronic administration of fluoxetine impairs neurogenic and endothelium-dependent relaxation of the rabbit corpus cavernosum smooth muscle.
(4) reported a successful treatment of a corpus cavernosum abscess with percutaneous aspiration and intravenous antibiotics in a paraplegic man on intracavernous papaverine injection therapy.
In the tissue of the corpus cavernosum, a significant downregulation of the miRNA-199 in the AD group and non-significant downregulation in the A and D groups compared to the C group were observed, whereas the miR-155 analysis demonstrated non-significant gene downregulation in the experimental groups compared to group C (Figure 4).
ESWT + MSCT Stimulates Angiogenesis and Vasodilatation in DMED Rats' Corpus Cavernosum. The result of Masson staining (Figure 2(a)) shows that the vascular smooth muscle of the DM + ESWT + MSCT group is obviously more, in comparison with vascular smooth muscle of the DM group.
Shou et al., "Phenotypic modulation of corpus cavernosum smooth muscle cells in a rat model of cavernous neurectomy," PLoS ONE, vol.
We have previously reported that older and diabetic rabbits showed impaired Ang-(1-7)-mediated relaxation suggesting that aging and diabetes-related erectile dysfunction (ED) may be partly due to decreased Ang-(1-7)-mediated relaxation of the corpus cavernosum [9].
Ginsenoids induced nitric oxide mediated relaxation of the rabbit corpus cavernosum Br.
Androgen regulates normal nerve function of the corpus cavernosum and helps to maintain the health of the endothelium (Alberson et al., 2012).
Role of nitric oxide and K+ channels in relaxation induced by polygodial in rabbit corpus cavernosum in vitro.
It is expressed in the corpus cavernosum and via its [AT.sub.1] receptor activates signaling pathways leading to vasoconstriction, proliferation, fibrosis, and oxidative stress that are thought to play a detrimental role in the progression of DMIED [7,11-13,15].
reported that TGF-b1 induced collagen synthesis in human corpus cavernosum smooth muscle and prostaglandin E1 (PGE1) suppressed this process.

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