Resistance

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Resistance

An effective upper bound on prices achieved because of many willing sellers at that price level.

Resistance Level

In technical analysis, a price that a security does not, or only rarely, rise above. Technical analysts identify a resistance level by looking at past performance. When the security approaches the resistance level, it is seen as an indication to sell the security, which will increase the supply, causing the security's price to fall back below the resistance level. If there are too many buyers, however, the security rises above the resistance level. When this occurs, the price of the security will likely continue to rise until it finds another resistance level. It is also called the overhead resistance level. See also: Price ceiling, Support (Support level).

resistance

An increased supply of a security. Compare support.

Resistance.

Resistance, or resistance level, is the top of a stock's current trading range, and the point at which the price is higher than investors are willing to pay. As stockholders sell at resistance level, the stock price goes down because supply exceeds demand.

For example, if, on a repeated basis, as stock A's price reaches $60, stockholders begin to sell, then $60 is considered its resistance level. But a trading range isn't fixed and investor attitudes change, so the resistance level tends to move higher or lower over time.

If stock A rises to $63 without a surge of selling, the current resistance line has been breached. This may be the result of a rising market or a bullish assessment of the stock's value. On the other hand, if selling increases at $57, that may become the new resistance level.

Conversely, the level at which demand exceeds supply and investors typically buy a certain stock is called support. It's the point that's considered the bottom of a stock's current trading range.

Technical analysts use the concepts of resistance and support in anticipating future stock price movements.

References in periodicals archive ?
Many risk factors associated with poor clinical outcomes in Fontan operation have been identified in clinical studies [611], which typically include high pulmonary vascular resistance, low ventricular ejection fraction, increased ventricular diastolic stiffness, and atrioventricular valve regurgitation.
Remember, hypoxemia can increase pulmonary vascular resistance aggravating PPH, and causing further dilation of the RV if this has not already occurred (Albert & Hague, 1997).
In July this year, the US Food and Drug Administration accepted for review Actelion's supplemental New Drug Application for Opsumit for the treatment of adults with inoperable CTEPH to improve exercise capacity and pulmonary vascular resistance.
In this context, the mechanism determining the increase of the pulmonary vascular resistance is not known, fact that can be clearly noticed in different studies.3-8 Several studies on Graves'disease show that the increase of pulmonary pressure depends on the increase of pulmonary vascular resistance a possible immune mechanism.5-8 The physiopathologic mechanism of producing pulmonary hypertension in hyperthyroidism is not clearly defined.
Nitrous oxide was avoided as it is known to increase pulmonary vascular resistance and heart rate.
We speculate that the absence of SERT activity leading to high levels of serotonin results in the maintenance of high pulmonary vascular resistance and low pulmonary blood flow seen during fetal life and contributes to the characteristic vascular abnormalities and pulmonary hypertension of ACD/MPV.
This oxygenated blood in the pulmonary circulation leads to pulmonary vascular dilatation, which in turns leads to decreased pulmonary vascular resistance, reduces septal displacement, and improves left ventricular function [15,16].
The objective of this study is to identify circulating miRNAs that (1) correlate with functional measurements of resistive afterload, (2) correlate with functional measurements of reactive afterload, and (3) can identify patients who respond to right heart catheterization (RHC) challenge conditions with at least a 20% decrease in pulmonary vascular resistance (PVR).
Acute adaptation of the RV to PH depends on both the stationary (pulmonary vascular resistance) and the pulsatile (PA stiffness, total pulmonary capacitance, and reflected wave) components of afterload [48].
All these lead to a significant increase in pulmonary vascular resistance, the consequence of which is pulmonary hypertension.
Vasodilators including ET-1 receptor antagonists, prostacyclin analogs, phosphodiesterase type 5 inhibitors, and rho-kinase inhibitors are common agents utilized to decrease pulmonary vascular resistance and to relieve PH.

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