In this study, which included a large representative cohort of patients hospitalized for NE during January 2000-December 2014, we identified nephrotoxic drug intake, visual disorder, microscopic or macroscopic hematuria,
leukocyte count >10 x [10.sup.9] cells/L, and thrombocytopenia ([less than or equal to] 90 x [10.sup.9]/L) as independent predictive factors of severe NE.
Leukocyte count at baseline and the end of therapy between groups (Table 3) was not significant (p = 0.772 and p = 0.178, respectively), although
leukocyte count after two weeks in combination therapy group decreased higher than in standard therapy group.
Total
leukocyte counts (TLC) were measured by automated hematology analyzer.
Leukocyte counts (eosinophils, monocytes, lymphocytes, neutrophils and basophils) were measured in whole blood drawn into 5 ml vacutainer tubes (Becton Dickinson and Company, New Jersey, USA) containing EDTA (40 [micro]L of K3-EDTA, 0.37 mol/L per tube).
Keywords: Acute appendicitis, Histopathology,
Leukocyte count.
Hambridge and Ogle reported a case of a 16-year-old male with involvement of the knee, shoulder, and ankle and a synovial fluid analysis significant for a
leukocyte count of 34,000 [11].
Total
leukocyte count was found significantly enhanced in diseased group as compared with control group.
A blinded, prospective trial concerning diagnostic value of
leukocyte count, neutrophil differential count and Creactive protein.
[1] Criteria for diagnosis of pancytopenia being hemoglobin less than 10 g/dL, total
leukocyte count less than 3,500/[micro]L, and platelet count less than 100,000/[micro]L.
Arthritis developed within two weeks after a single pristane injection which is assessed through total
leukocyte count. Total
leukocyte count was taken at day 0, 15 and 30 while lung tissue for histopathology was taken at the time of dissection (day 30).
Laboratory examination showed a
leukocyte count of 4.1 x [10.sup.3]/[micro]l and a serum C-reactive protein (CRP) level of 0.2 mg/dl.
The decrease in glucose concentration was directly proportional to both the
leukocyte count and preanalytical time, confirming unusually high leukocyte-mediated in vitro glucose consumption (3, 4).
