factor

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Related to EDHF: Endothelin, EDRF

Factor

A financial institution that buys a firm's accounts receivable and collects the accounts.

Factor

A third party that buys a firm's accounts receivable. If a firm is not confident in its ability to collect on its credit sales, it may sell the right to receive payment to the factor at a discount. The factor then assumes the credit risk associated with the accounts receivable. This provides the firm immediate access to working capital, which is important, especially if the firm has a cash flow problem. The price of factoring is determined by the creditworthiness of the firm's customer, not of the firm itself. It is also known as accounts receivable financing.

factor

A firm that purchases accounts receivable from another firm at a discount. The purchasing firm then attempts to collect the receivables.

factor

To sell accounts receivable to another party at a discount from face value. Thus, a firm in need of cash to pay down short-term debt may decide to factor its accounts receivable to another firm.

factor

  1. a firm that purchases TRADE DEBTS from client firms. See FACTORING.
  2. a firm that buys in bulk and performs a WHOLESALING function.
  3. an input (for example raw material, labour, capital) which is used to produce a good or provide a service.

factor

  1. 1a FACTOR INPUT that is used in production (see NATURAL RESOURCES, LABOUR, CAPITAL).
  2. a business that buys in bulk and performs a WHOLESALING function.
  3. a business that buys trade debts from client firms (at some agreed price below the nominal value of the debts) and then arranges to recover them for itself. See FACTOR MARKET, FACTORING.
References in periodicals archive ?
Under conditions in which the synthesis or action of NO is reduced, such as in atherosclerosis, hypercholesterolemia, or ischemia, PGI2 and/or EDHF release evoked by local mediators or pulsatile stretching may be a crucial compensatory or reserve mechanism for the maintenance of myocardial blood flow (30).
To our knowledge, this study is the first to demonstrate that EDHF participates in the acute relaxation response of coronary arteries to E2 in isolated perfused hearts from gonadectomized female and male rats.
Prostacyclin, EDHF, and potassium channels may have the most important roles as mediators of the relaxation response of E2 in gonadectomized and sham-operated male rats, whereas NO, EDHF, PGI2 and potassium channels may have important roles in sham-operated female rats.
Reduced EDHF responses and connexin activity in mesenteric arteries from the insulin-resistant obese Zucker rat.
In addition to NO and [PGI.sub.2], vasodilators of EDHF, sGC and HO are also involved in regulation of coronary artery dilation.
EDHF induced coronary artery dilation is related to potassium channel (Medhora et al., 2001).
EDHF and CO pathways partially participate in vasodilation induced by TGS in rat hearts.
These studies have indicated that in addition to and independently from their antioxidant effects, polyphenols enhance the productions of vasodilating factors; NO, endothelium-derived factor (EDHF), together with prostacyclin and inhibit the synthesis of vasocontrictor endothelin-1 in endothelial cells.
This vasorelaxant activity is caused by the stimulation of endothelial nitric oxide formation with the possible involvements of prostacyclin and EDHF activation as well.
In addition to NO, the endothelium is also able to release other relaxant factors, such as prostaglandin [I.sub.2] (PG[I.sub.2]) and EDHF (Mombouli and Vanhoutte, 1997).
Although several reports on plant extracts and constituents that affect the NO pathways and modulate the vascular contractility have been published (reviewed in Achike and Kwan, 2003), only a number of works signalizes a mechanism based on the release of EDHF (Schuldt et al., 2005; Kwan et al., 2004a, b; Villar et al., 2004).
In conclusion, in the present study we demonstrated that HSE induces a potent vasodilation in the rat superior mesenteric artery through a mechanism dependent on the production of NO, on activation of potassium channels and likely in the release of an EDHF. In addition, our results also demonstrate that the flavonoid rutin contributes for the vasodilator effect of HSE and points out the participation of other compounds in this effect.