cultures were scraped off and lysed in 50 [micro]M sodium phosphate buffer, pH 7.4.
These findings add to a growing literature that morphological transformation of astroglial cells
can take place in a pathway involving the plasminogen activating system and components of the focal cell adhesion machinery [15, 31, 32].
NGF content in culture medium of astroglial cells
was determined by ELISA using affinity purified anti NGF polyclonal antibody (Yabe and Yamada.
Furthermore, the present study revealed a significant correlation between initial stroke severity, assessed in terms of NIHSS values, and GFAP concentrations in ICH patients because the severity of clinical symptoms depends on bleeding volume (30), this supports the hypothesis that the GFAP release into plasma is tightly linked to the amount of astroglial cell
destruction caused by the expanding hematoma.
Particularly, in rat C6 astroglial cells
, it has been reported that TNF significantly increases chemokine Ccl-2 (monocyte chemoattractant protein-1), Il-6, iNOS (inducible nitric oxide synthase), and Mmp-9 (matrix metalloprotease-9) mRNA levels.
Further axonal collapse occurs because of native astroglial cells
participation and resides in head of the optic nerve and lamina cribrosa.
Recent evidence indicates that TNF[alpha] released by activated microglia is essential in inducing the neurotoxic A1 phenotype in astrocytes that lose their trophic function  and A1 astroglial cells
were found in autoptic tissues of patients with different neurodegenerative diseases, including ALS .
In addition, reactive microglia and astroglial cells
tend to generate iNOS resulting in the biosynthesis of relatively high quantities of NO .
Early reaction of astroglial cells
in rat hippocampus to streptozotocin-induced diabetes.
Stimulation of nerve growth factor synthesis/secretion in mouse astroglial cells
AQP4 is expressed in the astroglial cells
of kangaroo rat brain (Huang et al., 2001).
Propofol protects rat astroglial cells
against tert-butyl hydroperoxide-induced cytotoxicity; the effect on histone and cAMP-response-element-binding protein (CREB) signalling.