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When Issued

Referring to a conditional offer for a new security, subject to the security actually being issued. Before a new issue, underwriters canvass potential investors, who may or may not book an order to buy a portion the new issue. Orders made are said to be effective "when issued" because they may not be completed, especially if the offering is cancelled. Less formally, orders when issued are referred to as orders "with ice" or orders "when distributed."


Used on the consolidated tape to indicate that a stock is trading on a when-distributed basis: GY.WD 52.


Used in stock and bond transaction tables in newspapers to indicate that a security is trading on a when-distributed basis: ColgP wd.
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Cater MA, La Fontaine S, Shield K, Deal Y, Mercer JF (2006) ATP7B mediates vesicular sequestration of copper: insight into biliary copper excretion.
En el higado, el receptor ATP7B relacionado con la enfermedad de Wilson.
In Wilson's disease, copper is delivered to the liver but fails to be processed and transported to the gall bladder due to lack of or a defect in the ATP7B gene.
Wilson's disease (WD), also named hepatolenticular degeneration, is an autosomal recessive genetic disorder caused by defects of ATP7B gene.
Null mutation of the murine ATP7B (Wilson disease) gene results in intracellular copper accu mulation and late-onset hepatic nodular transformation.
WD is an autosomal recessive disorder caused by homozygous or compound heterozygous mutations in the ATP7B gene (ATPase, [Cu.
Se han descrito alrededor de trescientas mutaciones geneticas en diferentes grupos etnicos, la mas comun de las cuales es la sustitucion del aminoacido histidina por glutamato en la posicion 1069 del gen ATP7B.
Genetic analysis of ATP7B is the most decisive tool and is considered the only reliable test for family screening of asymptomatic siblings.
WD is caused by various mutations within the ATP7B (ATPase 7B, copper transporting protein/enzyme) gene.
Prevalent mutations in ATP7B have been identified in many population groups (http://www.