Rogers, "Neuronal expression of
tumor necrosis factor alpha in the murine brain," Neuroimmunomodulation, vol.
Polymorphic structure of the
tumor necrosis factor (TNF) locus: an NcoI polymorphism in the first intron of the human TNF-beta gene correlates with a variant amino acid in position 26 and a reduced level of TNF-beta production.
Monitoring the acute phase response: comparison of
tumor necrosis factor (cachectin) and C-reactive protein responses in inflammatory and infectious diseases.
Effects of pentoxifylline on
tumor necrosis factor production and survival during lethal E.
Some research points instead to a compound called
tumor necrosis factor alpha as a trigger for insulin resistance, Flier says.
The transcription factor NF-[kappa]B is a major regulator of immune responses stimulated by proinflammatory stimuli such as
tumor necrosis factor, viral and bacterial infections, and chemical and physical stressors.
Researchers at Ohio State University in Columbus injected rats with an immune-signaling molecule, called
tumor necrosis factor, that the body produces in response to illness.
In a study of 253 coal miners in France, NIEHS grantee Steven Kleeberger and colleagues investigated whether polymorphisms in two genes coding for the proinflammatory cytokines
tumor necrosis factor [alpha] (TNF) and lymphotoxin-[alpha] (LTA) modify lung response to oxidants.
In that time, concentrations of the AGE-linked cholesterol and the inflammatory compound
tumor necrosis factor alpha (TNF-alpha) also rose significantly, notes Crandall.
We found that pretreatment of human keratinocytes with PL inhibited SSR-mediated increase of
tumor necrosis factor (TNF)-alpha and also abrogated nitric oxide (NO) production.
The mouse ozone-sensitive marker genes encode toll-like receptor 4, an element involved in control of innate immunity;
tumor necrosis factor alpha, a cytokine involved in a number of inflammatory diseases; and myeloperoxidase, an enzyme involved in phagocytic cells' responses to immune stimulation.
Because treatment by tumor promoters [12-O-tetradecanoylphorbol-13-acetate (TPA), epidermal growth factor (EGF), or
tumor necrosis factor alpha (TNF[alpha])] transforms clone 41 cells to anchorage-independent and tumorigenic phenotypes, they are considered to be undergoing late-stage tumor promotion.