sell

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Related to L-selectin: E-selectin

Sell

To relinquish ownership of some asset in exchange for some monetary compensation. Selling may take any of several forms. In a cash sale, the seller receives cash or a cash equivalent immediately in exchange for the asset. In a credit sale, the seller surrenders ownership immediately in exchange for future payment, often with interest. An example of a sale is a simple transaction involving widgets. If the seller is willing to accept $2 per widget and the buyer wishes to purchase 100 widgets, then the seller gives to the buyer 100 widgets and in their place receives $200. See also: Buy.

sell

To dispose of an asset. Compare buy.

sell

see SELLING.
References in periodicals archive ?
Efficacy of the L-selectin antagonist was evaluated by measuring its ability to affect human lymphocyte trafficking in SCID mice.
L-selectin, one of the family members, is found on the surface of most leukocytes.
3- To investigate whether leukocytes from L-selectin tail serine-to-alanine knock-In mice exhibit impaired adhesion and migration in vivo.
Sulfated sialyl Lewis X, the putative L-selectin ligand, detected on endothelial cells of high endothelial venules by a distinct set of anti-sialyl Lewis X antibodies.
These results suggest that L-selectin mediates interactions with the uterus and that this adhesion mechanism may be critical to establishing human pregnancy," Fisher said, according to the Times.
When Fisher and researchers at a fertility clinic examined unused human embryos donated for research, they found that human trophoblasts make L-selectin.
L-Selectin has been defined as a neutrophil-rolling and lymphocyte-homing receptor (14,15).
The other molecules -- plasminogen activator inhibitor-1, E-selectin, L-selectin and P-selectin -- are often elevated in people with metabolic and cardiovascular disease.
Intercellular adhesion molecule-1 (ICAM-1) and E-, P-, and L-selectin are cellular adhesion molecules involved in the recruitment of leukocytes on the activated vessel wall during inflammation (1) and play an important role in the early stages of atherosclerosis and its complications (2).
L-selectin is a molecule which mediates the first step in this process -- the binding of neutrophils to the endothelial lining of blood vessels -- and the SMART Anti-L-Selectin Antibody is expected to interfere with this binding.
Following intravenous administration, Heparinase III cleaves HS binding sites for L-selectin, P-selectin, and chemokines that are expressed or released during ischemia/reperfusion, allowing them to be swept away in the blood stream.